KINESIOL 3U03 Lecture Notes - Lecture 5: Prednisone, Motor Control, Corpus Luteum
14 Apr 2016
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Document Summary
Autocrine: impact on cell that produces the hormone. Intracrine: efect on biological acivity in the cell. Paracrine: impact on cell that"s very close. Endocrine: hormone released into circulaion, all cells exposed to these, some cells will respond, while others will not. Igf-1: impact on own cells, causing liver cells to grow and develop, impact on other liver cells, endocrine hormone released into circulaion, allow us to achieve maximum potenial growth. Classic: pituitary, pineal, thyroid, thymus, parathyroid, testes, ovaries, adrenal, pancreas. Recent addiions: heart, stomach, liver, kidney, gi tract, adipose. Hormones act on issues remote from origin: released into circulaion to the receptor cell, all body issue exposure. Target issue: hormone receptor (cell wall/cytoplasm/nucleus) sensiivity, have a lock and key relaionship (lock: receptor, key: hormone) Protein: pituitary, pancreas, parathyroid, interact with cell surface receptor, fastest acing hormones. Cholesterol: testes, ovaries, adrenal cortex, cytoplasm receptors. Amine: adrenal medulla, thyroid, cell surface/nuclear receptors.
