PATH 3610 Lecture Notes - Lecture 2: Cardiac Output, Multiple Choice, Pharyngitis

• School: University of Guelph
• Department: Pathology
• Course: PATH 3610
• Professor: Andrew Vice

Document Summary

January 19, 2018 l6: circulatory system continued. If it develops in a vein, it will go to the lungs, preventing air flow. If gas and arterial, if it goes to one of the end-artery organs (brain, heart, kidney), it will cause an infarct, causing a heart attack, stroke, or will kill part of the kidneys. Shock: widespread hypoperfusion of tissues and cells resulting from reduced cardiac output and or reduced effective circulating blood volume, causes, hypovolemic. Identical in format to final: 60, summative, short to medium length questions, put the whole course together, facts and using them integration. Overall scheme in healing: stimulus injury, direct injury, damage-associated molecular patterns (damps, pathogen-associated molecular patterns (pamps) Local response: primitive response is phagocytosis and hypertrophy/hyperplasia. Inflammation reaction of blood vessels: repair, systemic response. Itis word: rhinitis, pharyngitis, tracheitis, bronchitis, rhino - nose, hepatitis, dermatitis, hepaticus - liver, pneumonia, pneumon - lung. Initial: minutes/days, major players plasma, neutrophils.