Kinesiology 3336A/B Lecture Notes - Lecture 2: Spasm, Heat Therapy, Ultimate Tensile Strength
10 Dec 2016
School
Department
Course
Professor
Document Summary
Body goes through a predictable sequence of healing. Our treatment/modality choice will be dependent on the phase of healing. Minimal ability to speed up this process, but interfering with this sequence will slow both recovery and return to sport. Cellular injury = altered metabolism + release of chemical mediators/proteins. Primary site damage at time of injury (immediately irreversible): we cannot do anything about this. Secondary site all the tissues close or adjacent to it, could be damaged. Damage by released proteins: damage as a result of body processes: Proliferative and regenerative healing leading to scar formation and repair to injured tissue. Fibroplasia begins within the first few days and the inflammatory signs should be decreased. Growth of endothelial capillary buds into the wound is stimulated by the lack of o2: these new growths allow increase o2 and blood flow. Body lays down type 3 collagen: weak tensile strength, beware initially very delicate scar but increase in collagen fibres strengthens it.
