SPHS-S 108 Lecture Notes - Lecture 15: Language Disorder, Stroke, Embolus
1 Aug 2018
School
Department
Course
Professor
Document Summary
Infarct: area of dead tissue resulting from blood flow interruption: anoxia. Infarct: tissue death: edema: swelling of brain tissue. Initial stroke effects (loss of consciousness, paralysis, etc. ) due to an edema. Initial effects most prominent in the first 3-5 days post-stroke: spontaneous recovery: neural reorganization, occurs when edema resolves or reduces in size improved blood flow, extent of spontaneous recovery depends on multiple factors, age, size/location of infarct. Causes of brain damage: head injury traumatic brain injury (tbi, (cid:862)ope(cid:374)(cid:863) or (cid:862)(cid:272)lose(cid:863, contusions/hematoma (bruising) Intracranial within the brain: meninges in the tissues surrounding the brain, lacerations (tissue tearing, diffuse axonal injury (damage to nervous tissues, growths neoplasms/tumors, occupy space, neural damage. Increase intracranial pressure: progressive deterioration, gradual loss of brain cells, dementia alzher(cid:373)er"s disease. Aphasia: acquired language disorder resulting from brain damage, usually to the left hemisphere, naming problems, fluency problems, auditory comprehension, repetition problems, sentence planning and production problems, agrammatism.
