FDNS 2050 Lecture Notes - Lecture 22: Overconsumption, Energy Density, Hyperplasia
14 Oct 2016
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Document Summary
Comparable to the set-point for temperature control. Instead of a set point for body temperature, there is a set point for body weight. Blood nutrient levels can send different signals to hypothalamus (glucostat; lipostat; aminostat) White fat tissue and muscle tissue metabolism. All activity except sleeping, eating, and sports-like exercise. The set-point theory: feedback control of energy intake & expenditure. Short-term (cid:373)echa(cid:374)is(cid:373)s (cid:373)ay or food i(cid:374)take. Peptides: cholecystokinin, obestatin terminate meal, ghrelin stimulates the appetite. Changes in blood glucose or amino acids. Increases in body fat will increase leptin release, decreasing the appetite. Decreases in body fat will decrease leptin release, increasing the appetite. The development of leptin resistance is thought to be a cause of obesity. May involve secretion of dopamine in the brain. Decreased energy expenditure will stimulate physical activity increased energy expenditure will induce physical inactivity. Increase in amount of fat per adipose cell. Maximal amount appears to be 1 mcg of fat per adipocyte.
