MEDRADSC 1B03 Study Guide - Midterm Guide: Virus Latency, Menopause, Senescence
14 May 2019
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Document Summary
Why do we age: aging is programmed genetically through cells, and limit"s reproductive capacity, predetermined cell death, wear/tear, cellular damage from accumulation of wastes, altered protein/lipid components. Increased collagen cross linkages: resident latent viruses, autoimmune reactions, environmental factors. Senescence- the time between old age and death. Lower testosterone levels: testes shrink, less sperm produced and less secretions of prostate, benign prostatic hypertrophy/hyperplasia (bph, male and female reproductive cancer later in life due to altered hormonal levels, prostate, uterine, breast. Lifestyle changes: diet, exercise, quit smoking, modify stress behaviour. Changes to intervertebral discs (absorb shock and reduce friction: herniation of the nucleus pulposus pressure on nerves, discs dry out/loss of elasticity loss of height. To maintain mobility and flexibility: regular moderate low impact exercise, good nutrition. Changes that reduce compliance/function: elasticity, rib movement (costal cartilage has calcified. Effects of reduced lung compliance: expansion for deep breathing and coughing expiratory volume, capacity and residual volume coughing ability risk of pneumonia.
