NUR240 Quiz: Chapter 30 Drug Chart
Document Summary
Blocks sodium ion channels in myocardial cells, reducing automaticity and slowing conduction of action potentials. Affects beta1 receptors in the heart and beta2 receptors in pulmonary and vascular smooth muscle. Do not combine with other antidysrhymics or anticholinergic drugs. Abrupt discontinuation may cause mi, severe htn, and ventricular dysrhythmias. Use cautiously in patients with diabetes because of the drugs hypoglycemic effects. Black box: prolonged administration can result in an increase in antinuclear antibodies; can cause a lupus-like syndrome in those taking the drug for more than a year. Patients with complete av block, severe hf, blood dyscrasias, myasthenia gravis. Monitor patients with serious cardiac disorders such as hf. Black box: abrupt withdrawal is not advised in patients with angina or heart disease. Patients with cardiogenic shock, sinus bradycardia, hf, copd or asthma (because it constricts smooth muscle in the airways) Blocks potassium ion channels and sometimes sodium ion channels. Resistant ventricular tachycardia that may be life threatening.
