PSY-250 Study Guide - Quiz Guide: Endocrinology, Sympathetic Nervous System, Stimulus Control

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PSY 250: Health Psychology
Exam 2 Study Guide
This guide recapitulates important lecture topics for each chapter. It also includes a list of textbook topics that warrant
further review. This guide should only be used as a study aid. It does not include (or make reference to) all exam
material. You are still responsible for all other lecture and textbook material.
Chapters 4 & 5: Health-Promoting & Compromising Behaviors
Exercise guidelines: Aerobic exercise (high intensity & long duration) is the recommended form;
individuals should engage in 30 minutes of moderate exercise each day and 20 minutes of vigorous exercise three
times per week.
Exercise vs. stress: Exercise only involves SAM axis activation while stress can activate both axes;
epinephrine may be metabolized differently and/or released in different amounts during exercise
Physical & mental benefits of exercise
Decreased risk of chronic illness
Weight control & increase in HDL levels (i.e., good cholesterol)
Effective coping strategy; can buffer against adverse effects of stress
Enhances immune functioning via several pathways (e.g., flushes bacteria out of airways, increases
circulation & effectiveness of antibodies & other cells)
Effects on mood (equally effective as SSRIs in treating depression)
Increases hippocampal & frontal lobe volume, which predicts enhanced cognitive functioning
Predictors of exercise:
o gender (men exercise more than women & this gap worsens with age);
o Habits of family & friends;
o high self-efficacy (reap more mood benefits & report less output/cost); convenience (i.e., number of
exercise facilities);
o low SES & minority neighborhoods and schools are disadvantaged due to less physical recess & lowered # of
facilities;
o importance predicts onset while perceived barriers predict maintenance
Risk factors for health-compromising behaviors:
low self-esteem, poor self-control, frequent interpersonal conflicts, deviance, peer pressure
Eating behavior
Weight is regulated around a set point via the ventromedial hypothalamus (governs the cessation of eating),
leptin (protein secreted from fat cells to decrease appetite), and ghrelin (substance secreted by stomach to
stimulate appetite)
Deviations from set point can produce changes in emotions, metabolic rate, and/or ghrelin release
Many people ignore their internal hunger signals and are motivated by other factors (e.g., visual cues)
Obesity: contributes to 40% of all cancers & many chronic illnesses; stress eating results in weight that is
distributed around the abdomen & is part of metabolic syndrome (i.e., set of symptoms that include high
LDL, low HDL, high triglycerides, hypertension) which predicts CVD, diabetes, hypertension, etc.
Windows of vulnerability for unhealthy eating: childhood since number of fat cells is determined early in
life and adolescence since the size of fat cells is still being determined; adolescence is window of
vulnerability for most health habits
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Eating disorders:
Common symptoms are body dissatisfaction & cognitive preoccupation;
Biopsychosocial factors include
genes,
serotonin,
self-esteem construal (overemphasis on body/appearance),
stress,
perfectionist personality (esp. for anorexics),
comorbid mental illness (depression, anxiety, OCD),
cultural pressure to be thin,
family issues (e.g., communication, mental illness including alcoholism)
BED (binge-eating disorder) is the most common; involves periods of excessive consumption that lead to
guilt; does not involve compensatory behavior
Anorexia involves self-induced starvation to the point where one is severely below weight; it is hard to treat
due to extensive level of distortion; high mortality rate
Bulimia involves bingeing (consuming thousands and thousands of calories in a short period of time and
then feeling guilty afterward) and purging (any compensatory behavior including using laxatives, vomiting, &
excessive exercise); usually are of normal weight or slightly overweight; better prognosis
Alcohol Abuse
Problem-drinking vs. alcoholism: only the latter involves true physical dependence (i.e., tolerance, cravings,
withdrawal symptoms, etc.)
Problem Drinking:
Consumption of large amounts of alcohol
Unable to cut down on drinking, even in the face of serious interpersonal & occupational
problems
Does not involve physical dependence
Alcoholism:
Involves physical dependence on alcohol (i.e., body has incorporated the substance into its
normal functioning)
Key features of addiction include tolerance, cravings, addiction, and withdrawal symptoms
Biopsychosocial model of alcoholism:
genes,
physical addiction (e.g., GABA, glutamate),
way of coping with stress,
short-term increases in mood and self-esteem,
drinking behavior of friends & family
comorbid mental illness (esp. social anxiety),
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adolescence
mid-late adulthood,
Comorbid mental illnesses
Anxiety disorders (e.g., GAD, Social, PTSD): GABA plays role
Bipolar disorder: drink during either (or both) phases
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Document Summary

This guide recapitulates important lecture topics for each chapter. It also includes a list of textbook topics that warrant further review. This guide should only be used as a study aid. It does not include (or make reference to) all exam material. You are still responsible for all other lecture and textbook material. Effective coping strategy; can buffer against adverse effects of stress. Enhances immune functioning via several pathways (e. g. , flushes bacteria out of airways, increases circulation & effectiveness of antibodies & other cells) Effects on mood (equally effective as ssris in treating depression) Problem drinking: consumption of large amounts of alcohol, unable to cut down on drinking, even in the face of serious interpersonal & occupational problems, does not involve physical dependence. Involves physical dependence on alcohol (i. e. , body has incorporated the substance into its normal functioning: key features of addiction include tolerance, cravings, addiction, and withdrawal symptoms, biopsychosocial model of alcoholism: