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PHGY 210 (301)
Ann Wechsler (112)
Lecture 12

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Department
Physiology
Course
PHGY 210
Professor
Ann Wechsler
Semester
Winter

Description
Lecture 12 Ventricular Tachycardia: very rapid sequence, PVC. These sequences are QRS signals that are not generated by the nodes, but are caused by the ventricular muscles. This is a signal of a clot in the heart, forcing blood into a ventricle. A ventricular tachycardia. Defibrillation uses a high current to force the heart to start working again, since this forces all heart cells to recalibrate itself, to become in sync again. Mapping of the Cardiac Electrical Activity: directly attach electrodes onto a heart, on the epicardial surface. The computer would then map of how the heart muscles activate by their order. Or using endocardial balloon inside of a heart to measure it. Circus movement re-entry is when an anatomical obstacle inside of the heart blocks the action potential from cycling through the heart. The CMR was discovered by Mines, who died in 1914 at the age of 27. Spiral-wave re-entry is when no obstacle is present, and the re-entry continues on in a spiral. Breakup of activation wave when tachycardia causes the action potential to split into multiple fronts. This leads to fibrillation. When shown on a ECG, multiple P waves are observed. Irregular heartbeat. Pulmonary vein ablation kills a ring of muscle cells around the pulmonary vein, which then prevents the atypical signals from the cells of the vein itself. Excitation -contraction Coupling: the sarcolemma is the cell membrane of muscle cells. Calcium inflow, binds to
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