MEDRADSC 2I03 Lecture Notes - Lecture 11: Chronic Obstructive Pulmonary Disease, Pulmonary Function Testing, Mediastinum
7 Dec 2016
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Document Summary
Mucous glands become hyperplastic (lots of mucous made) Swelling of mucous membranes of bronchi, spasm of smooth muscle in bronchial wall (narrowing of airways) increased response of tracheobronchial tree to allergens more mucus is made. Pets, pollen, molds, animals, fabrics, food, exercise, heat/cold, emotion. If untreated, there hard to breath (look at their jug notch) will be scarring hard to breath (look at their jug notch) Acini or terminal bronchioles undergo obstructive/destructiv e changes. Bulla burst and allows air in the pleural space (pneumothorax) lung collapse (atelectasis) Smoking, chronic bronchitis, air pollution, irritants of respiratory tract, anything that destroys cilia. Pt has bad colour (b/c bad gas exchange, heart tries to compensate by getting bigger) Injury to lung or parietal pleura that force air into the tissues of the chest wall. Permanently dilated large bronchi- elastic and muscular components of wall destroyed. Spontaneous or from chest trauma- could lead to pneumothorax.
