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ENH 220 (22)

Lymphatics System Diseases

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Environmental Health
ENH 220
Marcia Maguire

MYOSITISdamage to muscle cells from direct invasion by bacteria parasites viruses or fungi damages the sarcomere in the myofibril and results in inflammationthey are toxic products of bacteria clostridium perfringensalso called SEGMENTAL NECROSISCLOSTRIUDIUM PERFRINGENSGas Gangrene the strains of cp that cause food poisoning outbreaks also cause gas gangrene the spores of the cp found in soil on human skin in the intestine of healthy humans and animals Gas Gangrene is the gas produced by the bacteria thats growing in the tissues Clostridial Myonecrosis wounds can get contaminated with clostridia dead tissues supplies nutrients and anaerobic environment for growth of bacteriabacteria realease toxins such as alpha toxin which causes the red blood cells to break then destroy tissuePRGRESSION OF DISEASE toxins injure tissue usually large muscles thigh shoulder butt and diffuse into nearby healthy tissues and cause necrosis celldies uncontrolled or accidental the dead tissue serves as a site form growth of more clostridial bacteria toxin formation and gas production the necrotic tissue is black and filled with bubbles of gas always fatal if not treated Surgery and antibiotics neededCLOSTRIUDIUM TETANITETANUSLOCKJAW spores of clostridium tetani enter the wound ex Rusty nail wound must be necrotic and poorly supplied with blood bacteria produces neurotoxin and tetanospasmin which enters the spinal cord and interferes with neuroinhibitors that regulate the muscle contraction muscles released from inhibition uncontrollably contract clenching of jaw Arching of back flexion of arms death due to paralysis of respiratory muscles fatality rate between 10 to 70 head woundcan also have full recovery with no permanentdamage to the muscle NEONATAL TETANUS common in developing countries infected umbilical stump or circumcision with unsterile tools high death rate spastic paralysis of the paravertebral muscles which locks the back into a rigid arched positionTRICHINOSIS intestinal roundworm nematode parasite Trichinella spiralis most common larvae trichinae become encapsulated in the striated muscle acquired by eating rawnot enough cooked animal tissue that contains encysted larvae wormsin pork products larvae develop into adults in the small intestine and penetrate into blood stream then goes throughout the body larvae is encapsulated in the muscle tissuesSymptoms depends onof larvae ingested low death rate appears 8 to 15 days after depends onof parasites Vomiting and diarrhea sudden appearance of muscle soreness and pain swelling of upper eyelids or fever Eye pain retinal bleeding sweating chills and weakness In severe cases death can occur from myocardial failure
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