PAT 20A/B Lecture Notes - Lecture 12: Pleural Effusion, Cardiothoracic Surgery, Osteoporosis
22 Mar 2016
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Exercise: exercised induced asthma, ater, bronchospasm, pronounced when exposed to cold/dry air, to prevent. Respiratory infecions: increased inlammaion to tracheobronchial system -> increased hyperresponsiveness for 2-8 weeks. Nose & sinus problems: nasal polyps, r/t inlammaion of membranes, contribute to poor asthma control. Drugs and food addiives: asthma triad. Sensiivity to aspirin and nsaid (salicylic acid: oral/topic b-blockers cause bronchospasm, ace cause cough, sulphites (many foods and drinks, interfere with metabolic pathways = leukotriene producioning bronchonconstrictors. Gastroesophageal relux disease: thought that acid can enter lungs and cause bronchospasm, monitor esophageal ph and peak expiratory low rate. Emoional stress: lead to hypervenilaion and hypocapnia, causing airway narrowing, inluence varies. Pathophysiology degree of bronchospasm r/t inlammaion, hyper-responsiveness and exposure to endogenous/exogenous factors. Respiratory muscle funcion altered: altered venilaion, perfusion, arterial blood gas. Irreversible damage can result from remodelling and inlammaion airway remodelling. Wheeze, breathlessness, sensaion of chest ightness, cough, air trapping, hyperinlaion, dyspnea,
