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Lecture 7

NURS113 Lecture 7: Unit 7: Respiratory
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Department
Nursing
Course
NURS113
Professor
Alice Khin
Semester
Winter

Description
Chronic Bronchitis Emphysema Airway inflammation obstruction Enlargement of acini w loss of lung elasticity (distal to terminal bronchioles) Smoking, Air pollution Alpha 1 antitrypsin deficiency Bronchial wall thickened, Lumen narrowed Alveoli wall broken Proteases destroy alveoli Hypertrophy of mucous glands, mucus Compensatory hyperinflation produce bullae Productive cough 3 mo.yr. for 2 years SOB, cough, sputum Hypoxemia and cyanosis, polycythemia, Dyspnea, fish breathing pulmonary hypertension, cor pulmonale Barrel shaped chest Ventilationperfusion ratio mismatched Overventilate, maintain normal levels till late History of smoking Air hunger Coughing and frequent pulmonary infections Resp. failure, hypoxia, hypercapnia, respiratory acidosis Epiglottitis Acute inflammation of upper airway H. Influenza B Streptococci, Staph, pneumococci Croup Bronchiolitis Acute inflammation of bronchioles RSV, Influenza Bronchial Asthma Airway inflammation, bronchial hyperresponsiveness Acute intermittent Extrinsic (atopic) history of allergy, family history, skin test (+) Intrinsic (nonatopic) Respiratory infectionspsychological factors, severe, skin test () Type I hypersensitivity (IgE) NOCTURNAL (children, nonatopic) REFRACTORY (nocturnal, require cortisone injection) clinical triad (asthma, rhinosinusitis, nasal polyps) Bronchiectasis Permanent dilation of bronchi due to bronchial wall destruction Uncommon COPD Child infections (measles), lower resp. infection, TB, Obstruction (mucus, tumour) Emphysema affects alveoli and bronchiectasis affects main bronchi Hemoptysis, clubbing CF AUTOSOMAL RECESSIVE Fluid secretion in respiratory tractexocrine glands Mutation in CFTR gene (chromosome 7) Viscid mucus in bronchi, cause bronchiolitis, bronchitis and bronchiectasis after a while
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