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Class 12 February 5 Psych 275.docx

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Deanna Singhal

February 5, 2014 Seven steps in NT Action -1- NT molecules are synthesized from precursors under the influence of enzymes -2- NT molecules are stored in vesicles -3- NT molecules that leak from their vesicles are destroyed by enzymes -4- Action potentials cause vesicles to fuse with the presynaptic membrane and release their NT molecules into the synapse -5- Released NT molecules bind with auto receptors and inhibit subsequent NT release -6- Released NT molecules bind to postsynaptic receptors -7- Released NT molecules are deactivated by either reuptake or enzymatic degradation (or both with the exception of Acetylcholine which only undergoes degradation and has no reuptake.) Agonistic Drug Effects - Drug increases the synthesis of NT molecules o –e.g. by increasing the amount of the precursor - Drug increases the number of NT molecules by destroying the degrading enzymes - Drug increases the release of NT molecules from terminal buttons - Drug binds to autoreceptors and blocks their inhibitory effect on NT release - Drug binds to postsynaptic receptors and either activates them or increases the effect on them of NT - Drug blocks the deactivation of NT molecules by blocking degradation or reuptake Antagonist Drug Effects - Drug blocks the synthesis of NT molecules o –e.g. by
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