KNES 323 Lecture Notes - Lecture 11: Overtraining, Vasoconstriction, Osmotic Concentration
SchoolUniversity of Calgary
Course CodeKNES 323
ProfessorCarol Gibbons Kroeker
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(FOCUS ON THE STUDY GUIDES FOR SPECIAL TOPICS INSTEAD OF STUDYING
UNNECESSARY DETAILS FOR THE FINAL)!!!!!!!
Available forms of energy include: ATP (aerobic or anaerobic), plasma glucose,
glycogen in liver and muscle, gluconeogenesis, and fatty acids for diet/storage.
-Review metabolism for this unit.
Energy Systems for Exercise:
-For immediate energy production, phosphagen (phosphocreatine and ATP) is used.
4 ATP/min for about 5-10sec. (VERY short-term)
-Short-term energy production uses glycolysis. 2.5 ATP/min for 1-1.6 min.
-Long-term energy production is aerobic and gives 1 ATP/min for 2 mins and over.
Anaerobic vs. Aerobic Energy Systems:
-Anaerobic: ATP-PCR system for <10 sec. Glycolysis for <3 mins.
-Aerobic: Krebs, ETC, beta-oxidation (FATS!) for over 2 minutes!
-Aerobic is where fat burning kicks in.
Hormones During Exercise:
-Fat breakdown fatty acids
-Blood fatty acids increase during exercise.
-Cortisol, catecholamines and GH levels are high.
-Liver glycogen plasma glucose
-Glucagon, catecholamines and cortisol levels rise
-Non-muscle tissues shift to fat metabolism.
-Insulin secretion suppressed during exercise.
Regulation of Glucose Metabolism During Exercise:
-Glucagon rises, increase in liver glycogen breakdown
-Rise in epinephrine/norepinephrine, cortisol, GH, and thyroxine.
(Don’t remember all in detail)
Regulation of Fat Metabolism During Exercise:
-Rise in catecholamines increase in lipolysis
-Triglycerides fatty acids (using cortisol, epi/norepi, GH)
-Endorphins decrease pain response.
-Enkephalins increase strength considerably.
-Oxygen consumption increases and plateaus during exercise. It steadily drops of
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