ANP 1105 Lecture Notes - Lecture 10: Rh Blood Group System, Hematocrit, Viscosity

• School: University of Ottawa
• Department: Anatomy and Physiology
• Course: ANP 1105
• Professor: Jackie Carnegie

Document Summary

Blood clotting driven byy postivie feed back. Complicated process; no details about ht eintrinsic pathways under what conditions they two pathways are used, what are the goals of each pathway,solidifying the material. Blood (cid:272)lot (cid:373)ust ha(cid:448)e to (cid:272)oagulate agai(cid:374); as tissue repair e(cid:374)sues you do(cid:374)"t (cid:374)eed the (cid:271)lood (cid:272)lot anymore. Stop blood clotting just encase there was peeling of endothelial and exposure of collagen. This may results in blood vessel clotting not good. Getting rid of the factors that promote coagulation. The concentration that promote the fibrin threadmust be lowered. At a normal speed- spasming occurs slows down blood flow this makes clotting easier. Inacativating affects on the catalization of the pathway. If collagen is exposed it will activate blood clotting. Leeches want to keep blood free flowing; hirudin inhibits blood clot formation. Thrombus: clot that develops in a blood vessel, you want to try to restore blood flow. Plasmin in the clot dissolving enzyme,streptokinase treatment of thrombus . embolus.