PHS 4300 Lecture Notes - Lecture 7: Disseminated Intravascular Coagulation, Tissue Plasminogen Activator, Cardiac Arrhythmia

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Decrease flow to tissues by arteries: any decrease in supply of o2: hypoxia many reasons, anemia: decrease hemoglobin: blood loss, decrease iron, aterial obstruction: ischemia. Decrease blood flow from tussie veins. Leads to venous engorgement, edema, decrease removal of co2, wastes. Decrease flow of lymph (and blood flow: accessory system thin vessels, excess fluid/plasma diffuses btwn capillaries, and lymphatic vessels, plus key immune system role, blockage lymphedema. Picture blood doesn"t flow through lymph, plasma comes out of capillary walls and into tissues to be reabsorbed into lymphatic vessels (20:17- not all reabsorbed) Stationary blood clot in artery, vein, or on wall of heart: pathologically formed, not normal/physiologic homeostatic mechanism, made by of aggregated platelets, clotting factors, fibrin. Platelets tied together by fibrin and clotting factors: causes, atherosclerosis, after myocardial infarction (mi, ch18, damage to/replacement of heart valves, drugs, eg oral contraceptives, decrease venous blood flow, eg immobilization, shock. Immobilization can cause venous clots, can break off and spread.

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