BIOC32H3 Lecture 10: Lecture 10 - Synaptic Plasticity, Fatigue, & the NMJ

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periwinklerhinoceros137

29 Apr 2011

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Document Summary

Chapter 6 communication along and between neurons section synaptic plasticity p. Chapter 10 muscles and animal movement section motor control in arthropods p. 413. The modification of neuronal function as a result of experience. what we see in synapses is the activity of multiple axons synapsing on other axon terminals and not the postsynaptic membrane. Ca2+ levels will build on top of what is already there. a decline in the tendency to respond to a stimulus due to repeated exposure: i t can then stimulate the tail with electrical shock and then touch the gill = sensitization: increase in response to a gentle stimulus after exposure to a strong stimulus: one interneuron connected to both sensory and motor neuron as well as another facilitating interneuron. heterosynaptic facilitation: this slow chemical synaptic transmission occurs to deactivate a subtype of.

Related Questions

Botulinus toxin causes paralysis. It binds to presynaptic sitesat the motor end plate (neuromuscular junction) and prevents theexocytotic release of transmitter from vesicles. Predict theconsequences (increase, decrease, no change & explain why) ofblocking the release of acetylcholine at the neuromuscular junctionon the following: 1. Presynaptic resting potential 2. Presynapticaction potential amplitude 3. Presynaptic Ca2+ influx 4.Presynaptic transmitter release 5. Postsynaptic resting potential6. Postsynaptic end plate potential 7. Postsynaptic actionpotential amplitude 8. Postsynaptic action potential frequency 9.Muscle contractile strength