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PSYC12H3 (400)
Lecture

PSYC12H3 Lecture Notes - Blood Sugar, Insulin Receptor, Cardiac Muscle Cell


Department
Psychology
Course Code
PSYC12H3
Professor
Gerald Cupchik

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Question 1 - 657
Which of these proteins do not interact with RTKs?
Adaptors
Telomerase binding
Docking Proteins
Transcription Factors
Enzymes
Question 2 - 678
Mike is a patient of Dr. Grey. Mike was diagnosed with diabetes, and wants to know what is causing the lack of transport
of glucose throughout his body and organs. Dr Grey explains that…
There is a mutation in his PLC enzyme that refrains PLC from cleaving PIP2 into DAG.
There is a mutation in his PLC enzyme that refrains PLC from cleaving PIP2 into IP3.
There is a mutation in the adaptor protein IRS that binds to RTK.
There is a mutation in the docking protein IRS that binds to RTK.
There is a mutation in the transcription factor that binds to RTK.
Question 3 - 661
Which of the following is part of the process in closing the stomatal pore?
K+ influx channel closed and K+ efflux channel open
ABA causes calcium ions influx into the cell, signaling calcium release from the vacuole
K+ influx channel opened and K+ efflux channel closed
A and B
B and C
Question 4 - 670
Order the following steps of the events leading to the activation of a Insulin receptor. a.Trans-autophosphorylation occurs.
b.Conformational change in β subunits, kinase activation c. Insulin binds to α subunits. d.Insulin receptor substrate (IRS)
molecules phosphorylated.
b,c,a,d
c,d,a,b
c,b,a,d
d,b,a,c
None of above.
Question 5 - 667
In all the types of proteins listed below, which one(s) of them may interact with receptor tyrosine kinase (RTK)?
Adaptors
Docking protein
Transcription factor
Only two of the above are true
All of the above are true
Question 1 - 655
Which of the following is NOT a possible consequence of deleterious mutation in the genes for either the insulin receptor
or IRS2?
Diabetes type 1
Diabetes type 2
Cells stop responding to insulin
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