PSYC62H3 Lecture Notes - Lecture 7: Alcohol Dehydrogenase, Alcohol Withdrawal Syndrome, Blood Alcohol Content
21 Feb 2017
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Etoh and subjective experiences: alcohol leads to (lower bac) blood alcohol concentration. Etoh: adverse effects at high bac: alcohol stupor: dulled senses and poor cognitive function, blackout: reversible drug-induced dementia characterized by stupor and anterograde amnesia, alcohol poisoning: inhibition of autonomic system functions. Overwhelms medulla; body no longer has requisites to engage in life sustaining actions. Etoh: effects of chronic & heavy usage: cardiovascular system: Ischemia (decrease amount of blood oxygen), stroke, heart attack, cardiac arrhythmias: hepatic: (liver) Chronic disease of liver; degeneration of cells, inflammation and fibrous thickening of tissue (scarring) by alcoholism or hepatitis: fetal, nervous system: Combined with poor diet (thymine) lesions medial temporal lobes presents with unique neuropsychological impairments. Confabulations: creates a story for things they cannot explain, a way to (cid:498)fill in the gaps(cid:499) enzymes, acute tolerance: behavioral effects are weaker for declining bacs, metabolic tolerance: increase in liver alcohol dehydrogenase.
