CSB353H1 Lecture Notes - Lecture 3: Glucoside, Cysteine, Hydrolysis
28 Apr 2016
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Document Summary
Three main reasons for failure to infect plants. Plant species unable to support life-strategy requirements of pathogen (non-host) Plant has preformed structural barriers or toxic compounds that confine successful infection to specialized pathogen species (majority of non-host resistance) Pathogen attack recognition local and systemic signals resistance response. Lipophilic cuticula and trichomes contribute to reduce wettability of plant surface. Plants produce organic compounds that aren"t directly involved in growth secondary metabolites . May be stored as inactive precursors that are converted to active form following cell damage or exposure to enzymes. Eg. toxic compounds stored in vacuole; when pathogen enters cell, vacuole membrane ruptures, releasing contents. Eg. wounding increases myrosinase activity, which catalyze hydrolysis of glucosinolates, producing various bioactive toxic compounds. Avenacin a-1, a saponin, is a compound toxic to many fungi. > is produced in roots of oats, but not wheat. > g graminis var tritici (ggt) is extremely sensitive to avenacin a-1.
