LMP301H1 Lecture Notes - Lecture 1: Ammonia, Iron Poisoning, Impaired Glucose Tolerance
14 Sep 2016
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Equations: 95% interval = 2. 8 x [(analytical variance)2 +(biological variance)2, sensitivity (snout; catch all diseases), specificity (spin; catch all normals), positive predictive value, negative predictive value substances (normal -10 to 10, gfr 1/[pcr] ] ([h+] in nmol/l, pco2 in mmhg, [hco3. Decreased urine output most features normal edema possible. White blood cells/platelets (e. g. coagulation is currently happening) Due to loss of h+, alkali ingestion, or k+ deficiency (h+ lost in compensation) Body increases adh and aldosterone as it thi(cid:374)ks it"s lo(cid:449) i(cid:374) circulating water. Due to hyperventilation (usually acute with no renal compensation) Renal: atn (poisoning), protein deposition clogging the nephron, glomerulonephritis. Renal artery stenosis and nephrosclerosis glomeruli destroyed. End-stage renal disease (but if problem is in renal tubule (atn) then urine output remains normal) Pth (po4 becomes difficult, stays in body and combines with ca2+ to form crystals, pth secretion increased to make 1,25-oh vit. Sodium and water retention (leading to hypertension, feeding back to crf)
