Pathology 3240A Lecture Notes - Lecture 3: Antihistamine, Phalanx Bone, Trypsin
8 Oct 2016
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Document Summary
Need inflammation for life how body is programmed to act when injured/cells start dying (e. g. necrosis, infection) Even in tumours you have inflammation (i. e. there is necrosis in tumours) Dynamic: requires energy because it is an active process, starts with injury and culminates with healing/repair. Defense mechanism: but may become harmful. Non-specific and stereotyped: predictable similar reaction each time, regardless of nature of injury. The intensity, duration, and outcome is modified by variety of host and etiological agent factors. Ischemia (** very important cause: heart attacks, Red & swelling (rubor and tumour celsus) Loss of function of the inflamed part (functio l. virchow) Triangle because they are all interconnected & they happen very closely at the same time (dynamically active) This is acute inflammation: vessels: stage/site of inflammation, chemical mediators of inflammation, cells are the ones that know what to do and do it. Microcirculation is exhibited by all microvasculatures (veins, arteries, and capillaries)
