NURSE 305 Lecture Notes - Lecture 10: Deep Vein Thrombosis, Peripheral Edema, Captopril
Document Summary
A patient who has recently begun taking captopril (capoten) to treat hypertension calls a clinic to report a persistent cough. The nurse is preparing to administer an angiotensin-converting enzyme (ace) inhibitor to a patient who has hypertension. The nurse notes peripheral edema & swelling of the patient"s lips. The patient has a blood pressure of 160/80 mmhg & a hr of 76 bpm. Calcium channel blockers: dihydropyridines = doesn"t hurt pulse , amlodipine (norvasc, felodipine (plendil, nifedipine (procardia, adalat, non-dihydropyridines, verapramil (calan, diltiazem (cardizem, both classes -> arteriolar dilation -> decrease bp. Peripheral venous diseases: deep vein thrombosis (dvt) / venous thrombosis embolism, venous insufficiency. Peripheral arterial disease: the leading cause of pad is atherosclerosis from deposit of lipids within the vessel walls, common in diabetics & smokers, classic symptom of pad is intermittent claudication, complication of pad is necrosis.
