NUTR 384 Lecture Notes - Lecture 23: Skeletal Muscle, Diabetes Mellitus Type 2, Electrolyte
Document Summary
Type 1 diabetes: the pancreas does not produce insulin at all, complete dependence on exogenous insulin for glycemic control, often early onset @ a young age. Hypoglycemia: too little glucose, too much insulin, symptoms, diaphoresis, pallor, shakiness, jitters, altered mental status, confusion or disorientation, irritability, weakness, lightheadedness. Hyperglycemia: common complication, over time, leads to, retinopathy, neuropathy, nephropathy, symptoms, generally, develops quickly in type 1 diabetes, fatigue, excessive urination, excessive thirst, visual disturbance, unintentional weight loss. Dka (diabetic ketoacidosis: elevated levels of ketones with acidic blood ph, why, brought on by. Methods of glycemic coverage for type i diabetes. Insulin is currently the only treatment: basal or long-acting, short-acting, rapid-acting, intermediate-acting, usually given twice/day, at breakfast/dinner, pump, more common in younger patients, drip (acute setting only) Checking glucose: lab draws, clinical setting, bmp, finger sticks, q6h, tid before meals, hs, check on side on finger rather than middle, tid = 3 times/day, bid = 2 times/day.