ANSI 3543 Lecture Notes - Lecture 17: Maltose, Digestion, Lactose Intolerance
Document Summary
Moistens feed and disrupts structure: salivary amylase. Stomach: low ph denatures amylase, acid moistens cho and disrupts h-bonds, no cho digesting enzymes. Presence of cho in lumen stimulates amylase release. Hydrolyzes alpha-1,4 linkages: amylose maltose, amylopectin maltose + isomaltose (dextrin) Amylase hydrolysis of starch results in: starch, maltose, isomaltose, glucose. Enzymes found in brush border of duodenum. Brush border enzymes include: jejunum and ileum. Couples with na transport out of cell. Relative rates of absorption: active transport. Digestion in small intestine: similar to that of non-ruminant, cho digestion. Hindgut fermentation: similar to rumen, undigested cho and nfe. Small intestine: amylase, sucrose, maltase, lactase, 1,6-isomaltase. Hindgut fermentation: similar to rumen fermentation. Lactase deficient = lactose intolerant: genetic correlation, results in accumulation of gas and diarrhea. Diabetes mellitus: cells can"t absorb glucose due to lack of insulin, blood glucose levels increase and spill over into urine.
