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Lecture 18

WFSC 301 Lecture Notes - Lecture 18: Stomatitis, Fusobacterium Necrophorum, ZoonosisPremium

1 pages14 viewsSpring 2017

Department
Wildlife & Fisheries Sci.
Course Code
WFSC 301
Professor
Walter Cook
Lecture
18

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Tuesday, March 28, 2017 at 1415 PM
Lecture 18: Necrobacillosis
Etiologic agent: Fusobacterium necrophorum
Negative bacteria
Survives in the environment for week-> months
Can not penetrate intact skin
Geographic distribution:
Worldwide
Wildlife outbreaks in:
- Mule deer (CA)
- Elk (WY)
- WTD & pronghorn in Saskatchewan
- Caribou in Siberia
Transmission
Stress: overcrowding, heat and cold
Enter via cuts or damage to rumen/stomach/intestinal lining
Form of disease depends where the entry occurs
Causes:
• Footrot
Necrotic Stomatitis
Liver abscesses due to D. necrophorum
Signs:
fever + depressed
Oral: weight loss, swollen jaw or face
Feet: lameness, swelling between toas, localized tissue death,
swelling of joints and bones
Liver: may show any of the above or may lead to relatively rapid
death
Post morten findings and diagnosis:
Yellow/green necrotic ulcers and abscesses in affected area
Foul smelling
Can usually dianose it based on history, clinal signs
Human suceptibility to fusobacterium necrophorum
Humans may become infected and may have oral, liver and other
abscesses
They do not get infected from animals
Not considered zoonotic
But handle infected animals/carcasses with care
Impacts of Fusobacterium necrophorum
Not a widespread population limiting disease
Localized outbreaks can be substantial
Can be important in captive animals
Prevention and control
Minimize overcrowding, reduce stress, keep feed and water clean
Avoid producing muddy conditions (not always possible)
Can treat captive animals with anitbiotics, draining abscesses and
toe amputation
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