HES 250 Lecture Notes - Lecture 7: Asthma, Ephedrine, Pyruvic Acid
Document Summary
Research evidence: quality of sample, experimental control, statistically significant, majority of data, venues of publication, safety/side effects. Injection of oxygenated blood into an athlete before an event in an attempt to enhance athletic performance. Activates insulin receptors, thereby enhancing cellular responses to insulin and promoting anabolic responses. Transports fatty acids into mitochondria, thereby facilitating fat utilization. Reduce production of serotonin by blocking tryptophan from entering the brain and therefore delay the onset of fatigue. The central fatigue hypothesis: from rest to exercise: At rest: bcaa are sufficient to block free tryptophan from entering brain cells. During exercise: increased lipolysis causes an increase in plasma tryptophan because fatty acids displace tryptophan from its binding sites on albumin. Effect of cho and bcaa supplementation: exercise with cho. Cho supplementation inhibits lipolysis, therefore reducing plasma tryptophan (precursor to serotonin: exercise with cho and bcaa. Adding bcaa will block tryptophan from entering the brain, therefore reducing serotonin production.