KIN 155 Lecture Notes - Lecture 8: Cardiac Output, Sympathetic Nervous System, Vascular Resistance
7 Oct 2018
School
Department
Course
Professor
Document Summary
More fat gets to liver=more ldl produced. First liver produces vldls, which become ldls. Fat can go to muscle or store as body fat. More active, it goes to muscle; we absorb/use more fatty acids, expose liver less. Exercise can lower ldl, increase hdl= expose liver to less fat=good. Pa increase free fatty acid utilization, decrease blood triglycerides (decrease ldls, increase hdls)=less liver exposure. Evidence: male/female endurance athletes have 20-30% higher. Self report data, adjusted for age, alcohol, smoke, bmi. Each 100kcal/day expended was associated with 2. 09 mg/dl in hdl-c. Higher intensity activity associated with lower total cholesterol. Total energy expenditure is most important in cholesterol levels. Intensity pa is associated with primary prevention/treatment of mild hypertension. Population based epi study: just follow people, change nothing. Primary prevention: prevent hypertension from occuring at all. Blood pressure: measurement of force applied to artery walls. Harder the pressure=heart must work harder=require more oxygen. Hypertension: a factor of cardiac output x tpr.
