BIOL 2443 Lecture 8: L18

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20 Feb 2019
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Block 2: acetylcholine or ach, travels across the synaptic cleft, binds to membrane receptors on sarcolemma (motor end plate, causes sodium ion rush into sarcoplasm. Adp+pi (power stroke: myosin binds atp, releasing actin, myosin recocks using the energy from atp, hydrolyzing it to adp+pi (energized state) Ach release na+ channel activation . Block 2 bridge formation power stroke myosin reset/repeat . Contraction: neural stimulation of sarcolemma, causes excitation contraction coupling, cisternae of sr release ca2+, which triggers interaction of thick and thin filaments, consuming atp and producing tension. Ion pumps cease to function; ran out of atp: calcium builds up in the sarcoplasm. Block 2: myosin binds actin and contracts but cannot release (no more. Size of muscle and size of mvmt affect potential to reach peak of tension production: tension produced by whole skeletal muscles, depends on. Block 2: tension produced by whole skeletal muscles, recruitment (multiple motor unit summation)

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