BIO SCI E182 Lecture Notes - Carbonic Anhydrase, Osmotic Concentration, Central Pattern Generator

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2 Mar 2014

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Epo increases the number of red blood cells in the circulatory system, which carry oxygen. Epo would increase the red blood cell count, which would increase your oxygen carrying capacity. Riders would inject saline and plasma infusion to avoid detection of unusually high hematocrit. The line is much steeper in the bottom (not a straight line) Partial pressure of oxygen in a resting cell is 40 mmhg and 100 mmhg in alveoli. Binding of oxygen by hemoglobin is a reversible reaction needs to have a decrease in saturation level to unload the oxygen to the tissues. Effect of ph ph 7. 4 is considered normal (facilitates the unloading of oxygen from hemoglobin)-around 22 mmhg. If the ph is higher or lower than 7. 4, then this changes the binding of oxygen to hemoglobin. A lower ph (more acidic) would unload more oxygen. A higher ph (more basic) would unload less oxygen. At higher temperatures, oxygen unloading in the tissues is facilitated.

Related Questions

1. A blood vessel thatbecomes damaged and increases in diameter will fail because thetension in the walls _____ when compared to the previoulsyundamaged vessel.

A. decreases with anincreased diameter

B.decreases withpressure

C. increases with decreasedvessel thickness

D. decreases with increasedvessel thickness

E. increases with anincreased diameter

2. This nerve controls quiet respiration.

A. intercostalnerve

B. phrenic nerve

C. Diaphragmaticnerve

D.vagus nerve

E. pulmonaryplexus

3. Which of the following has no effect on oxyhemoglobindissociation?

A. lactic acid

B. fever

C. high tissue carbon dioxide

D. low ph

E. blood pressure

4. Surfactant of the lungs is created by

A. Oat cells.

B. lungmacrophages.

C. Type Ipneumocytes.

D. Type IIpneumocytes.

E. Goblet Cells

5. What is the medical termused to describe a lung that is no longer exchanging gases due tothe physical collapse of the alveoli (collpased lung)?

		A. apnea
		B.dyspnea
		C. acutebronchitis
		D. atelectasis
		E. asthma

6. If an individual removesthe surfactant from the lungs by inhaling and then exhaling asubstantial quantity of fresh water, the ____.

		A. alveoli would increase indiameter
		B. alveoli would decrease indiameter
		C. bronchioles would increasein diameter
		D. bronchioles would decreasein diameter
		E. vital capacity wouldincrease
		F. anatomical dead air spacewould increase

7. The chemoreceptors thatbest monitor the affects of respiration are found in

		A. the brainstem.
		B. the carotidbaroreceptor.
		C. the aoritcarch.
		D. the kidney.
		E. the cerbrospinalfluid.

8. Henry's Law deals with

		A. the total pressure of agas being made of the partial pressures of theindividual constituents
		B. the volume of a gas isrelated to the temperature and pressure
		C. the pressure of a gas isdirectly related to its velocity
		D. oxygen binding hemoglobinis temperature dependent
		E. oxygen binding hemoglobinis pH dependent

9. The amount of air in excess of tidal volume that can beinhaled with maximum effort is called

		A. vital capacity.
		B. inspiratory reservevolume.
		C. expiratory reservevolume.
		D. residualvolume.
		E. inspiratorycapacity.

10. Large amounts of air in the pleural spaces of a lung willcause the lung to collapse because ___________.

		A. the pressure of thetrapped air prevents inflation of the lung
		B. the trapped air will goout a hole called the pnuemothorax
		C. the lung is not longerstuck to the parietal pleural lining
		D. the lung has lostsurfactant
		E. the air will not beabsorbed outside of an alveoli

11. Most carbon dioxideproduced by cellular metabolism is

		A. carried by the red bloodcells bound to hemoglobin
		B. carried in the bloodplasma as bicarbonate
		C. carried in the red bloodcells as bicarbonate
		D. carried in the plamsa asdissolved gas
		E. carried in the red bloodcell as dissolved gas

12. If I have a blood vessel that doubles in diameter, at the samepressure I would expect about ____ as much blood to go throughit.

		A. 2 times
		B. 4 times
		C. 8 times
		D. 16 times
		E. 32 times
		F. 64 times

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Case 2 (7.5 pts)

R. S., a 55-year-old man, has been admitted to the hospital for treatment of acute gastrointestinal bleed. He is well known to the medical community for his chronic alcohol abuse and encephalopathy.

Past History: R. S. has been hospitalized four times in the past 24 months; most recently he was discharged 12 weeks ago for treatment of upper gastrointestinal bleeding. He has a 40-year history of smoking and drinks an unknown quantity of liquor daily. On previous admissions he has been treated for pancreatitis, alcohol withdrawal seizures, cirrhosis and associated ascites, coagulopathy, esophageal varices, anemia, gastrointestinal bleed, and gastritis.

Current Status: His current problems include hepatic encephalopathy, gastrointestinal bleed, coagulopathy, jaundice, history of tuberculosis (age 15 with treatment), and hypoxia; all present were on previous admission. Medications at last discharge included spironolactone (25 mg twice a day), ranitidine (150 mg twice a day), antacid (Maalox), and multivitamins.

R. S. was found unconscious in a pool of blood and vomitus. He was taken to the hospital and admitted to the medical intensive care unit. Initial vital signs were as follows: blood pressure 90/60, heart rate 112, and respirations 12. Intravenous infusion with normal saline was begun through a central line; oxygen was started at 3 L by nasal cannula. Physical examination revealed icteric sclera, clear lungs, grade Ill/TV systolic ejection murmur, and abdomen soft and without masses, liver percussed at 12 cm below the costal margin, and skin markedly jaundiced. Deep tendon reflexes were brisk and equal bilaterally.
Lab tests revealed the following: sodium 137 mEq/L, potassium 4.8 mEq/L, chloride 102 mEq/L, carbon dioxide 23 mEq/L, blood urea nitrogen 132 mg/dl, creatinine 1.3 mg/dl, glucose 115 mg/dl, hematocrit 27.7%, hemoglobin 9.3 g, white blood cell count 17,800 mm3, platelets 259,000 mm3, direct bilirubin 7.8 mg/dl, total bilirubin 16.4 mg/dl, amylase 42 somogyi units/dl, alkaline phosphatase 127 mu/dl, ammonia level 200 mg. Blood gases were as follows: Po.2 85 mm Hg, PCO2 30 mm Hg, pH 7.35, prothrombin time (PT) patient 17 sec., prothrombin time (PT) control 12.3 sec., partial prothrombin time (PIT) 46/36 sec.
Treatment with blood replacement was initiated; it included transfusion of 6 units fresh frozen plasma and 5 units packed red blood cells, with antacids, ranitidine, vitamin K, thiamine, and lactulose by nasogastric tube.
On the following day R. S.âs mental status was improved and his laboratory studies were as follows: sodium 133 mEq/L. potassium 3.5 mEq/L, chloride 105 mEq/L, blood urea nitrogen 15 mg/dl, creatinine 0.7 mg/dl, glucose 114 mg/dl, hematocrit 38.7%, white blood cell count 5200 mm3, PT 15.7/12 sec., ammonia 93 mg, alkaline phosphatase 92 m/dl. Vital signs were as follows: blood pressure 116/72, heart rate 72, respirations 15, afebrile.
On day 2 he was oriented X3 and was transferred from the intensive care unit. During the course of his hospitalization his total bilirubin ranged from 16.4 mg/dl down to 10.7 mg/dl at discharge.

What is the physiological benefit of having a hepatic portal system?

Does R.S have elevated bilirubin? If yes, does the portion of the total bilirubin that is direct indicate pre hepatic, post hepatic or an intrahepatic problem?

What is ascites? How do hypoalbuminemia and increased serum aldosterone levels contribute to the development of ascites?

Explain the cause of the patientsâ clotting defects.

The sclera of R. S.âs eyes are icteric and his skin yellowed. What is the cause of

these discolorations, and what do they indicate about his pathophysiology?

Explain why esophageal varices are the leading cause of death in patients with cirrhosis

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BIO SCI E182 Lecture Notes - Carbonic Anhydrase, Osmotic Concentration, Central Pattern Generator

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