HUN 4446 Lecture Notes - Lecture 26: Hyperglycemia, Low-Density Lipoprotein, Hypovolemia

• School: University of Florida
• Department: Food Science and Human Nutrition
• Course: HUN 4446
• Professor: Anne Mathews

Document Summary

Physiological disturbances for type 1 dm: absolute insulin deficiency, altered cho metabolism metabolism of fat ketonemia ketonuria ketoacidosis. Ketones have a low ph and cause the body"s ph to drop (that"s what ketoacidosis is: altered cho metabolism hyperglycemia glucosuria polyphagia polyuria. Polydipsia: altered protein metabolism muscle wasting, hyperglycemia, no insulin means to inhibition of the breakdown of nutrients (proteins/fats) Will have diabetes for the rest of life. Can cause someone to have very little muscle mass even though they"re. Insulin is an anabolic hormone that forces the uptake of overweight/obese: glucose, fatty acids, proteins, potassium. Hyperosmolar, hyperglycemis nonketotic coma: hypoglycemia (insulin shock/insulin rxn) Administer cho or glucagon: eat a simple carb that can be absorbed quickly, at least 15 g of carb, may use glucagon if someone cannot swallow if they"ve, diabetic ketoacidosis (dka) passed out. Bg > 250 mg/dl ph < 7. 25. Fluid and electrolyte replacement: hyperosmolar, hyperglycemic, nonketotic coma.