BIOL 3324 Lecture Notes - Lecture 23: Adrenergic Receptor, Adrenal Cortex, Adipose Tissue
Document Summary
Glucose levels are regulated by insulin: insulin decreases amount of circulating glucose, circulating fatty acids, circulating amino acids but increases protein synthesis and fuel storage. Glucose cells, more glycogenesis, less gluconeogenesis. Fatty acids adipose tissue, glucose adipose tissue. Glycogen synthesis in liver, more glycolysis: mannose, galactose, glucose can activate insulin producing beta cells, not stimulated by fructose. Alpha adrenergic inhibits: nor e stimulates alpha adrenergic, cck, glp-1, gip stimulate insulin secretion, incretins. Glucagon: made in alpha cells of pancreas, l cells of duodenum, synthesis is inhibited by glucose, to increase circulating glucose levels. You can tell if there are lots of aa circulating. Protein can be broken down for e. Generally conserved however if like relatively large amounts are there. Break down of nonessential globular protein: gluconeogenesis- make more glucose, glycogenolysis- break down glycogen, break down fat- via oxidation, lipolysis. Mineralocorticoids: aldosterone: makes aquaporins in dct, ct of nephron to concentrate urine and retain water.