HHP 3500 Lecture Notes - Lecture 46: Lipoprotein Lipase, Gastrin, Chyme
30 Nov 2016
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Document Summary
Most gi reflexes are stimulated by what is in gut, triggered by chemoreceptors, mechanoreceptors by stimuli: sitmuli are: distension of gi walls, chyme osmolarity, chyme acidity, and contents of chyme. This regulates luminal conditions to maximize nutrient digestion and absorption. Gi reflexes have nothing to do with nutrients currently in the blood, they just respond to presence of food in stomach. Regulation of gi response: regulated by nervous system (enteric ns) Submucosal nerve plexus-nerves regulate secretion activity into gut. Short reflexes-reflexes stay entirely with the enteric ns. Long reflexes-goes all the way to cns-contact with autonomic ns: integration in cns, response comes down then innervates enteric system to cause reflex. Feed-forward reflex-sight, smell, taste of food; emotions: regulated by endocrine system influence digestive processes. Endocrine cells scattered throughout the epithelium of stomach and small intestine. They sense contents of lumen, then release hormones into blood stream which then travels to gi and has effects.
