Psychology 1000 Lecture Notes - Lecture 7: Midbrain, Long-Term Memory, Basal Ganglia

• School: Western University
• Department: Psychology
• Course: Psychology 1000
• Professor: Dr.Mike

Document Summary

All work at level of the synapse, and can: Increase or decrease amount of transmitter: processes that terminate transmitter action, stimulates or blocks receptor sites. Cocaine: stimulates release of dopamine, prevents re-uptake of dopamine so that it stays in the synapse. Curare: blocks receptor sites for ach. Black widow venom: stimulates release of ach, movement and motor stimulus, heart races to the point of explosion. Botulism toxin: blocks release of ach. Nicotine: agonist stimulates release of drug effect, stimulates receptor molecules, duplicating effects of ach. Caffeine: antagonist prevents neurotransmitter from doing its job, blocks adenosine receptor sites, mimics adenosine in the adenosine receptors but makes you alert instead of sleepy (adenosine makes you sleepy) Epsp and ipsp are additive across space and time: graded potentials. At a given synapse, you can have all kinds of neurons connected. Generating hundreds of epsp and ipsp at different places across the nervous system. Raises threshold of next neuron in line after action potential.