KINE 2P97 Midterm: Lecture 4 Question Review

• School: Brock University
• Department: Physical Education and Kinesiology
• Course: KINE 2P97
• Professor: William Gittings

Document Summary

Products = 4 co2, 2 atp, 6 nadh, 2 fadh2, oxaloacetate. Citrate synthase increased activity with increased oxaloacetate; inhibited by atp, nadh. Isocitrate dehydrogenase allosterically activated by ca; inhibited by nadh + h. Ketoglutarate dehydrogenase activated by ca; inhibited by nadh + h & atp. Ketoglutarate dehydrogenase is said to be rate limiting; it is the closest to tca maximal flux. Ho(cid:449) (cid:373)a(cid:374)(cid:455) atp are produ(cid:272)ed per (cid:858)tur(cid:374)(cid:859) of the (cid:272)(cid:455)(cid:272)le? ( e(cid:373)e(cid:373)(cid:271)er to i(cid:374)(cid:272)lude redu(cid:272)i(cid:374)g equivalents nadh/fadh in the calculation) Inner mitochondrial membrane and its function is to convert h+ into atp. How does the etc convert the energy from electron transport into atp synthesis? (i. e. , Nadh 3 complexes, fadh2 2 complexes, skips complex i. Co(cid:373)ple(cid:454) iv: (cid:862)c(cid:455)tochro(cid:373)e c o(cid:454)idase(cid:863) tra(cid:374)sfers electro(cid:374)s fro(cid:373) c(cid:455)to. Hydrogen ions cross the inner membrane down concentration gradient (forms atp).