BIOM 3090 Study Guide - Final Guide: Benzodiazepine, Schizophrenia, Cannabinoid

193 views5 pages

Document Summary

Treat with: sympatholytics (clonidine, metoprolol, propranolol, prazosin, vasodilators (sodium nitroprusside, verapamil, ace inhibitors (enalapril, at1 antagonist (losartan, diuretics (hydrochlorothiazide, furosemide) Clonidine: an alpha 2 agonist, causes a decrease in ne in the cns decrease sns and increase. Propranolol: non-selective beta blocker decrease heart rate/cardiac output decrease in renin released (beta1 receptors in kidney) vasodilation **avoid in people with asthma. Metoprolol: selective beta blocker b1>>b2 same action as propranolol without the adverse effects of b2 blockage. Prazosin: alpha1 blocker decrease in arterial/venous tone vasodilation decrease co (na+ and water retention occurs as compensation) Sodium nitroprusside: no causes and increase in cgmp vasodilation; only used in emergency situations negative effects and possible cyanide poisoning (metabolites) Verapamil: ca2+ blocker inhibits ca2+ release associated with smooth muscle contraction relaxation vasodilation (also affect in heart muscle decrease hr) Enalapril: ace inhibitor cannot convert at1 to at2 (no vasoconstriction; vasodilation as result); bradykinin is no longer inhibited vasodilation.