PSYC 102 Study Guide - Midterm Guide: Leptin, Cholecystokinin, Ghrelin

• School: UBC
• Department: Psychology
• Course: PSYC 102
• Professor: Mark Lam

Document Summary

Ve(cid:374)t(cid:396)o(cid:373)edial hypothala(cid:373)us: (cid:862)satiety (cid:272)e(cid:374)te(cid:396)(cid:863: lesion = fat, stimulate = skinny. Late(cid:396)al hypothala(cid:373)us: (cid:862)hu(cid:374)ge(cid:396) (cid:272)e(cid:374)te(cid:396)(cid:863: lesion = skinny, stimulate = fat. G(cid:396)ehli(cid:374): ho(cid:396)(cid:373)o(cid:374)e i(cid:374) the sto(cid:373)a(cid:272)h se(cid:374)ds (cid:862)hu(cid:374)ge(cid:396)(cid:863) sig(cid:374)als to the hypothalamus: appetite increases. Leptin: hormone released by fat cells to inhibit hunger: appetite decreases. Cholecystokinin = hormone that counteract the effects of ghrelin and decreases hunger. Value that establishes a range of body and muscle mass we tend to maintain. When we eat too little, we drop below our set point, regulatory mechanisms kick in to increase our appetite or decrease our metabolism. In this way bodies defend against weight loss. Theory holding that obese people are motivated to eat ore by external cues than internal cues. Can be born with more fat cells. Lower metabolic rates at which their bodies burn calories. With less sensitivity to leptin than thin people.