01:146:328 Study Guide - Midterm Guide: Trematode Life Cycle Stages, Paragonimus, Watercress
29 Dec 2019
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Egg (lateral spine)>freshw ater stimulates hatching (cilia of miracidium beat rapidly and cause rupture)>mira cidium(infects snail)>sporoc yst(daughter sporocysts)>c ercaria>dh. Egg (terminal spine)>freshw ater stimulates hatching (cilia of miracidium beat rapidly and cause rupture)>mira cidium(infects snail)>sporoc yst(daughter sporocysts)>c ercaria>dh. Egg (small, lateral knob)>freshw ater stimulates hatching (cilia of miracidium beat rapidly and cause rupture)>mira cidium(infects snail)>sporoc yst(daughter sporocysts)>c ercaria>dh. Cca-cholangi ocarcinoma, bile duct damage, toxic parasite secretions. Liver rot by juveniles (acute fascioliasis, migration to liver necrosis, hepatomegaly-s welling of liver) Necrosis of lung tissue (coughing up blood, ectopic sites) Live in blood vessels, non-operculated eggs, no redia stage, no metacercaria sage, only trematode not foodborne, no pharynx bc they don"t feed on solid food. Cercarial dermatitis: cercariae penetrate skin and cause allergic reaction. Egg entrapment: get stuck in gut/bladder wall, swept into bloodstream leading to liver/spleen; granulomatous response host immune cell surround the egg due to sea being released from miracidium and build into granuloma/fibrous tissue kills miracidium brain, skin)
