PHYL 1010Y Chapter Notes -Catabolism, Ketone, Glycosuria

• School: Dalhousie University
• Department: Physiology
• Course: PHYL 1010Y
• Professor: n/a

Document Summary

Fasted state metabolism: liver glycogen becomes glucose, adipose lipids become free fa and glycerol that enter the blood, glycerol converted to glucose in the liver, fa released into the blood and used for e by other tissues (like muscle, muscle glycogen can be used for e, glycogen becomes pyruvate or lactate, then made into glucose in liver (gluconeogenesis, muscles also use fa and break down their proteins to aa that enter the blood, brain can use only glucose and ketones for e. Pancreatic hormone that decreases plasma glucose concentration: either increase transport into cells or increases metabolic use of glucose, increases glycogen synthesis, aerobic metabolism of glucose, protein & triglyceride synthesis, anabolic hormone that promotes glycogen, protein & fat synthesis, targets liver, muscle and adipose tissue, decreased glucose plasma shuts off insulin release, when deficient/absent, cells go into catabolic metabolism.