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Department
Philosophy
Course
PHIL 320
Professor
Jennifer Johnson
Semester
Fall

Description
Week 10 lecture: Schizophrenia Somatoform & dissociative disorders – neuroses. Change in term to psychoses in the 1980’s. Thought to be harder to address using things like counselling as more serious break from reality. Because occurs cross culturally, think strong biological underpinning. History: Dementia Praecox (biological) -> Split mind (functional perspective) -> Splitting in Schizophrenia involves a breaking down of thought processes. Everything still functions – comp. to dissociative identity disorder – parts that don’t function. Counselling mostly unsuccessful. Meds can help. Positive symptoms: functional excess. Negative symptoms: functional deficit. Disorganised symptoms can be considered positive symptoms – depends on approach. Some patients experience visual hallucinations (role of other drugs?). Apathetic Avolition – also evident in basal ganglia disorders Inappropriate affect (disorganised symptom?) Catatonia – basal ganglia/dopamine? Damage to dopamine system/ prefrontal cortex? No longer inhibiting or selecting. V heterogeneous condition. Originally thought that paranoid subtype had a better prognosis. Speech tends to be incoherent. Odd motor symptoms – could this be hinting at underlying pathology? Echolalia – repeating what is said to them Echopraxia – repeating what is done to them Hints of problems in childhood – eg. don’t smile as much as other children -> schizoi
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