PHPR 2823 Study Guide - Final Guide: Aldosterone, Fumaric Acid, Glomerulonephritis

• School: Lipscomb University
• Department: PHPR - Pharmacy Practice
• Course: PHPR 2823

Document Summary

Conceptual model of ckd: kidney disease outcomes & quality initiative (kdoqi, kidney disease: improving global outcomes (kdigo) Initiating factor causes nephron loss compensatory nephron hypertrophy and glomerular htn proteinuria causes tubular cell damage. Initiating both associative and causative: causes nephron loss, advanced age, systemic inflammation. Low renal mass: dyslipidemia, progressing mechanisms causes further decline in, racial minority. Progressing mechanisms: diabetes [t2dm >t1dm, hypertension, glomerulonephritis, other, hyperglycemia, hypertension, proteinuria, smoking, obesity. Umbrella term meaning inflammation of the glomerulus: diffuse all glomeruli, focal- some but not all. Segmental local parts of individual glomerulus: numerous etiologies. Symptoms of ckd: asymptomatic, vital to screen high risk dm and htn, edema, worsening htn, fatigue (anemia, ca/vitamin d abnormalities, renal osteodystrophy, electrolyte abnormalities, hyperphosphatemia /hyperkalemia, high risk: screened annually. Ckd screening: dm, htn, autoimmune diseases, and many more. Spot albumin/scr ratio: scr & gfr calculation, blood pressure, questions for therapy affects drugs we use.