NSG 4413 Lecture Notes - Lecture 54: Vital Signs, Cardiac Output, Perfusion
13 May 2019
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Document Summary
A clinical syndrome resulting from inadequate tissue and organ perfusion. Cells consume approximately 25% of the oxygen delivered. Stage 1 = compensatory mechanisms are effective in maintaining relatively normal vital signs and tissue perfusion. Stage 2 = compensatory mechanisms begin to fail; metabolic and circulatory derangements become more pronounced. Laboratory studies = lactate, abg, cbc, cmp, pt, ptt, and h&h. Oxygen, monitor vital signs, labs, respiratory status, mentation, and urine output. Chest pain, hypotension, cool skin, oliguria, decreased mentation, dyspnea, and crackles. Loss or disruption of sympathetic tone causes peripheral vasodilation and subsequent decreased tissue perfusion. Allergic reaction to speci c antigen > food, insects, or medication. Assess for known allergens and obtain through history. Clinical manifestations = generalized erythema, uticaria, pruritus, anxiety, and restlessness, dyspnea, wheezing, chest tightness, a warm feeling, nausea and vomiting, angioedema, abdominal pain, and laryngeal edema. Life threatening = epinephrine, uids, steroids, bronchodilators, and vasopressors. Initiated by an infection in the bloodstream > bacteremia.
