NURS 3331 Chapter Notes - Chapter 17: Adrenergic Agonist, Adrenergic Receptor, Angina Pectoris
Document Summary
The most common mechanism by which drugs activate peripheral adrenergic receptors. Produce their effect by binding to adrenergic receptors and mimicking the actions of natural transmitters. Act on terminals of sympathetic nerves to cause ne release. By blocking ne reuptake, drugs can cause ne to accumulate within the synaptic gap. Drugs that inhibit monoamine oxidase (mao) can increase the amount of ne available. Contain a catechol group and an amine group. A benzene ring that has hydroxyl groups on two adjacent carbons. Acts on beta 1 and beta 2 receptors. Therapeutic applications and adverse effects of adrenergic receptor activation. To understand the effects of any specific adrenergic agonist you need: The identity of the receptors at which the drug acts. The arrest of bleeding, , which alpha 1 agonists support through vasoconstriction. Stop bleeding in the skin and mucous membranes. Caused by dilation and engorgement of blood vessels in the nasal mucosa. Congestion is relieved by alpha 1 mediated vasoconstriction.
