BIOL126 Lecture Notes - Lecture 6: Respiratory Alkalosis, Respiratory Acidosis, Aspirin Poisoning
Document Summary
Acid-base balance (3 marks: describe the effects of hypo- and hyper-ventilation on pco2 levels and their relationship with ph, hypoventilation (breathing slow) Ventilation inadequate to meet metabolic needs for o2 delivery and co2 removal. Affects acid-dbase balance elevated production of h+ Ge(cid:374)eratio(cid:374) of e(cid:454)(cid:272)ess h+ i(cid:374) this (cid:449)a(cid:455) leads to a (cid:858)respirator(cid:455) a(cid:272)idosis(cid:859: hyperventilation (breathing fast) hypocapnia: below normal co2 in arterial blood. Rate of (cid:448)e(cid:374)tilatio(cid:374) e(cid:454)(cid:272)eeds the (cid:271)od(cid:455)(cid:859)s (cid:373)eta(cid:271)oli(cid:272) (cid:374)eeds for co2 re(cid:373)o(cid:448)al. Arterial pco2 levels drop mainly due to anxiety, fever and aspirin poisoning. Alveolar po2 increases during hyperventilation and arterial po2 therefore increases. As hb is almost fully saturated, very little extra o2 is added to the blood. Decreased production of h+ leads to a (cid:858)respirator(cid:455) alkalosis(cid:859: dis(cid:272)uss the (cid:272)auses of (cid:858)(cid:373)eta(cid:271)oli(cid:272)(cid:859) a(cid:374)d (cid:858)respirator(cid:455)(cid:859) a(cid:272)idosis a(cid:374)d alkalosis a(cid:374)d the (cid:271)od(cid:455)(cid:859)s compensatory mechanisms, and interpret acid-base data. Respiratory acidosis: results from excessive levels of co2 in body fluids and hypoventilation.
