EXSS2028 Lecture Notes - Lecture 13: Exercise Intensity, Norepinephrine, Oxidative Phosphorylation

• School: University of Sydney
• Department: Exercise and Sport Science
• Course: EXSS2028
• Professor: Kieron Rooney

Document Summary

O2 uptake (vo2) response to exercise: exercise onset, lag in vo2 o2 deficit (so tap into other energy sources e. g. glycolysis, oxidative phosphorylation in mitochondria is not. Immediately achieved: delayed response in energy supply, anaerobic energy systems to supply the required energy, lag of vo2 because o2 supply does not meet demand; so there is a delayed response in energy supply. Immediate energy systems provide atp (pcr: exercise protocol implemented in figure = constant rate steady-state exercise protocol, trigger for oxidative phosphorylation = lots of substrates adp + pi. Acute muscle adaptations: energy sources for muscle as a function of activity duration. Increase rate of oxidative phosphorylation = lass of mass action = you exercise & demand more atp due to inadequate adp + pi. Why the delay in o2 uptake kinetics: at the onset of exercise, there is inadequate adp & pi to turn on oxidative phosphorylation.