LSB111 Lecture Notes - Lecture 4: Pulmonary Edema, Ventricular Fibrillation, Reperfusion Injury

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13 Aug 2018
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Lecture 4 cardiovascular disorders: pathway of blood flow (brief, right atrium right ventricle lungs left atrium left ventricle . If there is loss of myocytes (e. g. infarction or atrophy: decrease force of contraction. If there is an increase in myocytes (e. g. hypertrophy) Increase force of contraction: heart rate, controlled by autonomic nervous system, sympathetic nerves (adrenergic activity) Impulses from baroreceptors sent to cardiovascular control center. Increase in co by heart (increase in hr and contractibility) by sympathetic nervous system activity: vasoconstriction of blood vessels by sympathetic nervous system activity. Increase co and resistance returns blood pressure to homeostatic range. Ventricular fibrillation: region contracts in uncoordinated manner, medical emergency, atherosclerosis progressionn, angina or myocardial infarction (mi, angina. If severe leads to acute heart failure within hours (cardiogenic shock: progressive/congestive heart failure (late complication, arrhythmias, pericarditis (inflammation of the pericardium, can lead to cardiac tamponade, ventricular aneurysm.

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