PSYC-275 Lecture Notes - Lecture 8: Scotopic Vision, Retinotopy, Retina

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Mid-Term 2 Review – Part 1 (Modules 4.2 & 4.3)
What are totipotent, pluripotent, multipotent, and unipotent cells?
1.
Describe the stages in the induction of the neural plate (know the
difference between the plate, groove, tube, and crest). Which parts
develop in to the CNS and PNS?
2.
What is neural proliferation and where does it occur?
3.
What is the difference between radial and tangential migration?
4.
What factors facilitate the migration and aggregation of the
developing neurons?
5.
What are the Chemoaffinity Hypothesis and Topographic Gradient
Hypothesis? Describe the experiments that supported each notion?
6.
What is synaptogenesis? What type of cells mediate this process?
7.
What are the determinants of neuronal (and axonal) survival?
8.
What is the difference between apoptosis and necrosis? Which of
these is activated in early development? Why?
9.
What is the effect of neuronal death and synaptic rearrangement on
the selectivity of synaptic transmission?
10.
What structural changes are responsible for post-natal brain
growth?
11.
What are the effects of experiences such as musical training, exercise,
enriched environments?
12.
What is adult neurogenesis? Where does it occur?
13.
What is a brain tumor? Describe the main (distinctive) properties of
the four types discussed in class?
14.
What are cerebrovascular disorders?
15.
Midterm 2 review
Tuesday, March 6, 2018
9:38 AM
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What are cerebrovascular disorders?
15.
What is the difference between an infarct and a penumbra? What
factors influence the fate of the cells within the penumbra?
16.
What is the difference between cerebral hemorrhage and ischemia?
17.
List and define the 3 ways in which a cerebral ischemia can develop.
18.
Describe the toxic cascade the develops due to cerebral ischemia.
19.
What is the difference between a contusion and concussion?
20.
Define the term “encephalitis”. Describe the properties of the 2
bacterial and viral infections discussed in class.
21.
What is toxic psychosis?
22.
What is tardive dyskinesia?
23.
Why are neurological disorders of genetic origin almost always
caused by abnormal
recessive
genes?
24.
What is epilepsy? Describe the 2 main types and their subtypes.
25.
What is the difference between anterograde, retrograde, and
transneuronal degeneration?
26.
Why is axon regeneration possible in the PNS but not in the CNS?
27.
Describe the “2-Stage Model” of neural reorganization after brain
damage. (Hint: collateral sprouting & release of inhibition)
28.
What is denervation supersensitivity?
29.
What is Phantom Limb Syndrome? How is treated using the “mirror-
box therapy”?
30.
Discuss the 3 long-term treatment & rehabilitation interventions
discussed in class.
31.
Mid-Term 2 Review – Part 2 (Chapter 5)
Chapter 5
What is the “law of specific nerve energies”?
32.
What do mean when we say “visible” light when referring to the
electromagnetic waves?
33.
List the names and functions of the structures of the human eye.
34.
Identify the different properties of the center and the periphery of the retina.
35.
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Document Summary

Mid-term 2 review part 1 (modules 4. 2 & 4. 3) Describe the stages in the induction of the neural plate (know the difference between the plate, groove, tube, and crest). What are the chemoaffinity hypothesis and topographic gradient. List and define the 3 ways in which a cerebral ischemia can develop. Describe the toxic cascade the develops due to cerebral ischemia. Describe the properties of the 2 bacterial and viral infections discussed in class. Describe the 2 main types and their subtypes. Describe the 2-stage model of neural reorganization after brain damage. (hint: collateral sprouting & release of inhibition) Discuss the 3 long-term treatment & rehabilitation interventions discussed in class. Mid-term 2 review part 2 (chapter 5) List the names and functions of the structures of the human eye. Identify the different properties of the center and the periphery of the retina. Rods are most abundant in the periphery of the retina. However, even within the periphery there is differential distribution.

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