Practical Nursing PNP301 Lecture Notes - Lecture 6: Globin, Iron Overload, Polycythemia

• School: Seneca College
• Department: Practical Nursing
• Course: Practical Nursing PNP301
• Professor: m

Document Summary

Less blood reaches the various organs: decreased cell function, mild acidosis develops. It is estimated about 600,000 canadians are living with heart failure. It is on the rise as more people survive heart attacks and other acute heart conditions: www. heartandstroke. ca. Sns stimulation (cid:894)co(cid:373)pe(cid:374)satory(cid:895: release of epinephrine/nor-epinephrine. Myocardial hypertrophy: walls of heart thicken to provide more muscle mass stronger contractions. Pathophysiology of hf: renal perfusion/ juxtaglomerular apparatus se(cid:374)ses (cid:858)hypo(cid:448)ole(cid:373)ia(cid:859, kidneys release renin which converts, angiotensin i angiotensin ii (compensatory) which causes: Aldostero(cid:374)e release na/h20 retention (via adh secretion) As the compensatory mechanisms continue over time, they can worsen heart failure. Excessive tachycardia: vasoconstriction: the resista(cid:374)ce agai(cid:374)st (cid:449)hich heart has to pu(cid:373)p (cid:894)i. e. , (cid:859)s afterload(cid:895), a(cid:374)d (cid:373)ay therefore co, na and water retention: fluid (cid:448)olu(cid:373)e, (cid:449)hich preload. If too (cid:373)uch (cid:862)stretch(cid:863) (cid:894)d/t too (cid:373)uch fluid(cid:895) stre(cid:374)gth of contractio(cid:374) a(cid:374)d co. Excessive tachycardia diastolic filli(cid:374)g ti(cid:373)e (cid:448)e(cid:374)tricular filli(cid:374)g v a(cid:374)d co.