PSY 1205 Lecture Notes - Lecture 1: Traumatic Brain Injury, Stroke, Amnesia
Document Summary
Develops over short time period; fluctuates in severity. Dementia (memory impairment) & deterioration of other cognitive functions. Onset of cognitive deficits due to cerebrovascular accident(s) Typically occurs abruptly & follows step-wise progression with rapid decline. Deterioration of brain tissue in frontal & temporal lobes. Specific deficits vary with injury site(s), age, etc. Involved substance or medication capable of producing the neurocognitive impairment. Course of neurocognitive deficits consistent with timing of substance use. Lewy bodies: abnormal protein deposits in nuclei of brain cells disruption of processes involving memory and motor control. Parkinson"s disease: deterioration of substantia nigra & dopamine deficiency. Majority of patients will develop major ncd / dementia. Neurocognitive disorder due to huntingdon"s disease: huntingdon"s disease: inherited degenerative disease. Formation of abnormal clusters of prion molecules infection brain damage major ncd. Creutzfeldt-jacob disease: formation of small cavities in the brain.
